For years, it has been an accepted dogma that obesity led to type 2 diabetes by causing inflammation, which was perceived to change the way the body responds to the effects of insulin. Scientists theorized that chronic, low-grade tissue inflammation related to obesity contributed to possible insulin resistance. However, new study suggests that this theory may need to be updated, the Endocrine Web reports.
Researchers from Children’s Hospital in Boston found out that inflammation is actually responsible for activating two different proteins that play key roles in keeping blood sugar levels stable. Without the presence of inflammation, these two proteins remain dormant.
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